Amebic liver abscess: 1966–1976
Identifieur interne : 003935 ( Main/Exploration ); précédent : 003934; suivant : 003936Amebic liver abscess: 1966–1976
Auteurs : J. Marc Shabot [États-Unis] ; Marcel Patterson [États-Unis]Source :
- The American Journal of Digestive Diseases [ 0002-9211 ] ; 1978-02-01.
English descriptors
- Teeft :
- Abdominal pain, Abnormal, Abnormal liver scan, Abscess, Alkaline phosphatase, Amebiasis, Amebic, Amebic abscess, Amebic liver abscess, Amebicidal therapy, Amoebic liver abscess, Case report, Chloroquine, Chloroquine phosphate, Chronic disease, Clinical picture, Clinical suspicion, Diffusion test, Digestive diseases, February, Gamma globulins, Hepatic, Hepatic abscess, Hepatic lobe abscess, Indirect hemagglutination, Intestinal amebiasis, Laboratory findings, Large defect, Liver abscess, Liver scan, Lobe, Metronidazole, Metronidazole therapy, Mexican nationals, Needle aspiration, Night sweats, Open catheter drainage, Physical examination, Pleuritic pain, Positive diffusion, Positive serology, Presumptive diagnosis, Previous experience, Pulmonary changes, Pulse rate, Quadrant, Race distribution, Relapse, Resolution time, Right chest, Right hepatic lobe, Scan, Serum albumin, Serum bilirubin, Unknown medications, Weeks emetine, Weeks metronidazole, White blood cell count.
Abstract
Abstract: A 10-year retrospective analysis of 15 patients with amebic liver abscess is reviewed and represents a continuation of the previous 2-decade experience at our institution (37 patients). Records were studied to determine the population affected, presenting symptoms, physical and laboratory findings, type and response to therapy. Most patients were rural males of lower socioeconomic status in the third to fifth decade of life. They presented as a febrile illness with abdominal pain for an average of 2 1/2 months duration. Significant physical abnormalities were tender hepatomegaly (93%), right-sided pulmonary changes (40%), and fever (66%). All patients had abnormal liver scan, positive amebic serology, and hypoalbuminemia. All patients promptly responded to amebicidal therapy except one whose therapy was delayed. Clinical suspicion, liver scanning, serologic testing, and response to therapy are the keys to diagnosis.
Url:
DOI: 10.1007/BF01073184
Affiliations:
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Marc Shabot</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><placeName><region type="state">Texas</region></placeName><wicri:cityArea>Division of Gastroenterology, Department of Medicine, University of Texas Medical Branch, 77550, Galveston</wicri:cityArea></affiliation></author><author><name sortKey="Patterson, Marcel" sort="Patterson, Marcel" uniqKey="Patterson M" first="Marcel" last="Patterson">Marcel Patterson</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><placeName><region type="state">Texas</region></placeName><wicri:cityArea>Division of Gastroenterology, Department of Medicine, University of Texas Medical Branch, 77550, Galveston</wicri:cityArea></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Digestive Diseases</title><title level="j" type="abbrev">Digest Dis Sci</title><idno type="ISSN">0002-9211</idno><idno type="eISSN">1573-2568</idno><imprint><publisher>Kluwer Academic Publishers-Plenum Publishers</publisher><pubPlace>New York</pubPlace><date type="published" when="1978-02-01">1978-02-01</date><biblScope unit="volume">23</biblScope><biblScope unit="issue">2</biblScope><biblScope unit="page" from="110">110</biblScope><biblScope unit="page" to="118">118</biblScope></imprint><idno type="ISSN">0002-9211</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9211</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Abdominal pain</term><term>Abnormal</term><term>Abnormal liver scan</term><term>Abscess</term><term>Alkaline phosphatase</term><term>Amebiasis</term><term>Amebic</term><term>Amebic abscess</term><term>Amebic liver abscess</term><term>Amebicidal therapy</term><term>Amoebic liver abscess</term><term>Case report</term><term>Chloroquine</term><term>Chloroquine phosphate</term><term>Chronic disease</term><term>Clinical picture</term><term>Clinical suspicion</term><term>Diffusion test</term><term>Digestive diseases</term><term>February</term><term>Gamma globulins</term><term>Hepatic</term><term>Hepatic abscess</term><term>Hepatic lobe abscess</term><term>Indirect hemagglutination</term><term>Intestinal amebiasis</term><term>Laboratory findings</term><term>Large defect</term><term>Liver abscess</term><term>Liver scan</term><term>Lobe</term><term>Metronidazole</term><term>Metronidazole therapy</term><term>Mexican nationals</term><term>Needle aspiration</term><term>Night sweats</term><term>Open catheter drainage</term><term>Physical examination</term><term>Pleuritic pain</term><term>Positive diffusion</term><term>Positive serology</term><term>Presumptive diagnosis</term><term>Previous experience</term><term>Pulmonary changes</term><term>Pulse rate</term><term>Quadrant</term><term>Race distribution</term><term>Relapse</term><term>Resolution time</term><term>Right chest</term><term>Right hepatic lobe</term><term>Scan</term><term>Serum albumin</term><term>Serum bilirubin</term><term>Unknown medications</term><term>Weeks emetine</term><term>Weeks metronidazole</term><term>White blood cell count</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Abstract: A 10-year retrospective analysis of 15 patients with amebic liver abscess is reviewed and represents a continuation of the previous 2-decade experience at our institution (37 patients). Records were studied to determine the population affected, presenting symptoms, physical and laboratory findings, type and response to therapy. Most patients were rural males of lower socioeconomic status in the third to fifth decade of life. They presented as a febrile illness with abdominal pain for an average of 2 1/2 months duration. Significant physical abnormalities were tender hepatomegaly (93%), right-sided pulmonary changes (40%), and fever (66%). All patients had abnormal liver scan, positive amebic serology, and hypoalbuminemia. All patients promptly responded to amebicidal therapy except one whose therapy was delayed. Clinical suspicion, liver scanning, serologic testing, and response to therapy are the keys to diagnosis.</div></front></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Texas</li></region></list><tree><country name="États-Unis"><region name="Texas"><name sortKey="Shabot, J Marc" sort="Shabot, J Marc" uniqKey="Shabot J" first="J. Marc" last="Shabot">J. Marc Shabot</name></region><name sortKey="Patterson, Marcel" sort="Patterson, Marcel" uniqKey="Patterson M" first="Marcel" last="Patterson">Marcel Patterson</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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